248) eled virus infected differentiated FBBC-1 cells as evidenced by the production of in |
249) unctionally assess actively proliferating cells as well as slowly dividing cells, in |
250) recall responses for interferon-γ+CD8+ T cells as well as to the establishment of s |
251) High PD-L1 expression (>10% cells) as well as low TCR diversity (≤64 |
252) optosis considerably in gastric carcinoma cells, as shown by flow cytometry, and had |
253) cerebellum was employed to sparsely label cells for subsequent morphological analyse |
254) vel approach for the generation of CAR-NK cells for targeting SARS-CoV-2 and its var |
255) ium influx and gap junctions on pyramidal cells for the modulation of SPW-Rs in a co |
256) engineered stable 3D/2D perovskite solar cells for their commercial utilization. |
257) d more efficient production of engineered cells for therapeutic applications, includ |
258) or generating 'off-the-shelf' S309-CAR-NK cells for treatment in high-risk individua |
259) tabolic state of rare, early-activated T cells, we adapted mass cytometry to quanti |
260) In both airway and immune cells, we found an association between RSV |
261) In HCC cells, we found that expression of HBx sup |
262) in vitro assays with P-gp overexpressing cells, we observed enhanced accumulation o |
263) tors that are highly selective for cancer cells, we optimized the amino acid moiety |
264) Using the human granulosa-like KGN cells, we show that this effect is not the |
265) The adenocarcinoma cells can be categorized into different su |
266) lungs or the intestine, where epithelial cells can be infected with virus. |
267) Alternatively, donor T cells can become IFN- γ/IL-17 cytokine ex |
268) S309-CAR-NK cells can specifically bind to pseudotyped |
269) Furthermore, S309-CAR-NK cells can specifically kill target cells e |
270) s functions, their expression in B and T cells is strictly segregated throughout ev |
271) that radiation dose to circulating immune cells is critical for tumor control. |
272) cellular protein released from bacterial cells is directly proportional to the conc |
273) However, whether the metabolism of immune cells is dysregulated in the TME by cell-i |
274) samples are unavailable or the number of cells is scarce. |
275) Cancer cells treated with C108 or cancer cells wi |
276) ility, and this effect was potentiated in cells treated with CYP1A1 siRNA. |
277) gland dysfunction, primary parotid gland cells treated with PGE2 have increased c-J |
278) nal load is induced in BRCA1-complemented cells treated with a PARPi. |
279) ntitative proteomic study of G1 phase ALL cells treated with vincristine. |
280) 021 that described dysregulated miRNAs in cells or animals infected by SARS-CoV-2 or |
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