254) Brain aging is associated with molecular, cellular, and structural change |
255) ding of many aspects of brain function at molecular, cellular, and system levels and |
256) evelopment of targeted therapies based on molecular characteristics is a promising a |
257) ted topics concern new information on the molecular characteristics of gliomas (infr |
258) was identified for its morphological and molecular characteristics where the (Inter |
259) The molecular clock, an intrinsic transcriptio |
260) f Per2, one of the core components of the molecular clock, at both mRNA and protein |
261) α, a negative regulator of the circadian molecular clock, is pro-convulsant through |
262) rmation on age-dependent changes in their molecular composition are scant. |
263) hotosynthesis, but the amount, timing and molecular composition of released carbon a |
264) Moreover, their molecular composition reflects the physiol |
265) The molecular diagnoses included biallelic var |
266) ith extensive locus heterogeneity and the molecular diagnoses made are for emerging |
267) re they can come to fruition in providing molecular diagnoses to genetically unsolve |
268) se genetic disruption yields very similar molecular effects as treatment of cells wi |
269) tanding sex differences in behavioral and molecular effects of stress has important |
270) ants, KTN1 mRNA expression, putamen GMVs, molecular effects of substances, and SUD, |
271) e subgroups to stratify patients based on molecular features of disease, clinical ch |
272) pluripotent cells (PSCs) recapitulate key molecular features of human PSCs with grea |
273) The molecular features of promising photosensi |
274) Molecular genetic analyses reveal major al |
275) h lifestyle factors, but the interrelated molecular genetic pathways are incompletel |
276) Finally, we discuss the importance of molecular genetic testing for precision me |
277) ophores, notably including its outlook on molecular imaging in vivo. |
278) demonstrate a tunable scaffold for the PA molecular imaging of protease activity wit |
279) heart failure can be made via the use of molecular imaging techniques, including po |
280) rks interface at the target site, where a molecular initiating event (MIE) occurs an |
281) ry drug (NSAID) bioactivation is a common molecular initiating event for hepatotoxic |
282) metabolites is commonly regarded as a key molecular initiating event. |
283) ated the expression, biological function, molecular mechanism, and clinical implicat |
284) including the tissue and cell mechanism, molecular mechanism, and drug delivery. |
285) etable QSAR models and reveal the related molecular mechanism, we performed a system |
286) es that enhanced our understanding of the molecular pathology of gynecological cance |
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