281) Currently, whether immunoresponsive gene 1/itaconate axis exerts a modulatory |
282) regulatory effect of the immunoresponsive gene 1/itaconate axis has been recently do |
283) te for the deficiency of immunoresponsive gene 1/itaconate axis led to enhanced micr |
284) Our results suggest that gene co-expression is highly plastic and t |
285) ted genes in burn scar tissue by weighted gene co-expression network analysis (WGCNA |
286) es in the Young (TEDDY) study to generate gene co-expression networks. |
287) Gene coexpression networks based on the hu |
288) Next, two distinct methods of weighted gene coexpression network analysis (WGCNA) |
289) After aggregation of gene coexpression networks based on the hu |
290) Gene duplications of KU70 have the hallmar |
291) functional diversification after repeated gene duplications. |
292) urred through multiple, relatively recent gene duplications. |
293) Gene ontology (GO) and Kyoto Encyclopedia |
294) Gene ontology analysis of the budesonide-m |
295) After clustering and gene ontology analysis, 23 molecular marke |
296) ase of the cell cycle, and it modulates a gene signature associated with DNA deamina |
297) TCGA has recently revealed that a hypoxia gene signature is associated with poor UM |
298) This gene signature was validated in an indepen |
299) h carried human AML-ETO or MLL-AF9 fusion gene to establish haplo-identical and majo |
300) the miR-21 binding site on exon 4 of GAS5 gene to generate a GAS5 mutant abolished i |
301) rn a C-terminally tagged copy of the PF16 gene to the original locus, which resulted |
302) France Genomics and Ethics Network (UK-FR GENE), which has been set up to reflect on |
303) ing to the formation of the fused EWSFLI1 gene, which codes for an aberrant transcri |
304) entified a copy of IS6110 within the moaX gene, which turned out to be specific for |
305) Gene-ontology analysis revealed the role o |
306) lly relevant populations, undetectable by gene-expression analysis. |
307) Mutations in the ABCA4 gene are a common cause of Stargardt disea |
308) or SK4 channels and encoded by the KCNN4 gene, are activated by a rise of the intra |
309) paRG did not activate PXR in the reporter gene assay, illustrating the limitations o |
310) ) HepaRG cells, as well as a PXR reporter gene assay, were used to investigate the m |
311) All those who took part had changes in a gene called ALK, which is involved in cell |
312) a certain resistance mutation in the EGFR gene, called T790M. |
313) Paralogs of the Obp50 gene cluster are expressed in metabolic an |
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