313) GABAA receptor α-subunit is the dominant molecular determinant of drug selectivity. |
314) GABAA receptor α-subunit is the dominant molecular determinant of drug selectivity. |
315) estimated 90% of CHD cases, but so far, a molecular diagnosis remains unsolved in up |
316) Mendelian genomic analyses, a presumptive molecular diagnosis was made in 13/27 (48% |
317) maging (MSI) enables the visualization of molecular distributions on complex surface |
318) ore been expanded to the 3D realm whereby molecular distributions within a 3D sample |
319) Molecular docking, western blotting, flow- |
320) argets of DHA, and these were verified by molecular docking, qRT-PCR, and Western bl |
321) ess, the structural translation of ligand molecular efficacy into G protein activati |
322) ields single point measurements of ligand molecular efficacy. |
323) Here, we used molecular, genetic, and pharmacological ap |
324) valuable model organism for studying the molecular, genetic, and tissue-level mecha |
325) Determining the molecular identity and the pharmacology of |
326) In both instances, the receptors' molecular identity has not been fully defi |
327) Molecular investigations revealed that Dic |
328) e a platform to advance hypothesis-driven molecular investigations of nsCL/P and oth |
329) the promise of high-frequency, real-time molecular measurements in complex sample m |
330) al aptamer-based sensors enable real-time molecular measurements in the living body. |
331) Breakthroughs in molecular medicine have positioned the amy |
332) ur discipline emerged at the forefront of molecular medicine with the rapid uptake o |
333) of pathologic diagnoses, and to validate molecular methods for gene expression stud |
334) en investigated using culture-independent molecular methods. |
335) Molecular monitoring is recommended monthl |
336) After TKI reintroduction, molecular monitoring is indicated every 2 |
337) functional contexts, reflecting specific molecular pathways, cell types, circuits, |
338) roducts have identified many cellular and molecular pathways, providing crucial insi |
339) A next-generation gas chromatograph-molecular rotational resonance (MRR) spect |
340) ∼2 K, resulting in a limited number of molecular rotational and vibrational level |
341) ly provide further proof for the possible molecular signaling basis of the antimetas |
342) understood, EOAT may demonstrate specific molecular signaling patterns. |
343) tand the conditions under which different molecular signals are employed by microgli |
344) cal mediators of synaptic plasticity, the molecular signals underlying this process |
345) roups, suggesting the sulcus has a unique molecular signature compared to the neighb |
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