340) ure to E-ALF shows lesser transcriptional response, with most of the M . |
341) educed EC50 for ATP and increased maximal response at human P2X7. |
342) hree patients had a pathological complete response at surgery (27.3%). |
343) invasive meningiomas elicited a monocytic response at the tumour-brain interface. |
344) yielded two empirically derived metrics: response bias (log b) and task discriminab |
345) king are known to differ substantially in response bias and accuracy, and these diff |
346) althy participants consistently develop a response bias in favor of the rich alterna |
347) t downstream genes, so-called cooperation response genes (CRGs). |
348) Type 1 interferon response genes (ISGs) were among those tha |
349) In network modules that contain immune response genes associated with T1D, we obs |
350) cterial EVs in modulating cellular immune response, the challenges and recent advanc |
351) For a two-activity level response, the error rates on the ankle, hi |
352) evated levels of type 1 IFN with cellular response, the persistence of cross-present |
353) ursor, induces a strong immunostimulatory response through a newly identified UU-con |
354) umidity, and precipitation influence host response through alterations in vitamin D |
355) despite triggering a robust innate immune response through the activation of cytopla |
356) meostasis were activated while the T-cell response were relatively repressed in thos |
357) s the delayed-type hypersensitivity (DTH) response were significantly decreased. |
358) remission), or treatment response, were included, as well as studie |
359) m (ER) lumen triggers an unfolded protein response (UPR) for stress adaptation, the |
360) The unfolded protein response (UPR) is an intracellular signali |
361) chieves accurate predictions of treatment response across classes of antidepressants |
362) Despite variability in induction response across hepatocyte donors, the fol |
363) ter regulator of the antioxidant cellular response, although it is currently conside |
364) d healing as they reduce the inflammatory response, although the bacterial load is n |
365) hondrial dysfunction, and an inflammatory response are factors contributing to IRI-r |
366) abnormal type I and III interferon-gamma response are thought to be responsible. |
367) ductions are achieved for specificity and response bias, and in other domains. |
368) pondence between anhedonic phenotypes and response bias, the PRT has been reverse-tr |
369) f community input were used to reduce the response burden on Indigenous communities. |
370) ives and reduce the disease and treatment response burden. |
371) tre and an enhanced neutralising antibody response compared to monomeric RBD. |
372) akinra had opposite effects on the immune response compared to standard of care trea |
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