352) owever, it remains unclear which specific genes expressed in which layers of the gro |
353) ts show that interactions between SNPs in genes from the aging pathways influence su |
354) ne coexpression networks based on the hub genes from the candidate modules were then |
355) We selected candidate genes from well-known aging pathways (IGF1 |
356) that CCR2, LMO7, STEAP4, NNAT, and TCF7L2 genes had good diagnostic performance for |
357) th male mice, and 26 of the 35 BA-related genes had marked gender difference in mice |
358) Almost all identified genes had not previously been implicated i |
359) only a small number of disease-associated genes have been identified. |
360) nknown significance" (VUS) in transporter genes have not been characterized. |
361) 246 AD risk genes have not been identified as AD risk |
362) G pathway enrichment reanalysis of the 21 genes identified five key genes (CCNB1, BU |
363) in ten emerging recurrent NDD-associated genes identified from large scale sequenci |
364) a confirm previously reported PrCa target genes identified through GWAS/eQTL overlap |
365) howing cannabis-associated alterations at genes important for early development. |
366) e transcriptional network, normalizes the genes important for hepatocyte function, a |
367) network and normalizes the expression of genes important for hepatocyte function, i |
368) regulate expression of the corresponding genes through CRISPR interference (CRISPRi |
369) rogen receptor-coregulatory and essential genes through mRNA 3' UTR sequence complem |
370) edge about Mendelian phenotypes and their genes through the years. |
371) lead to the suppression of cilia-related genes while also inducing nucleolin. |
372) istant alleles or non-functional effector genes while they can still be effectively |
373) on the number of TRBV subgroups and TRBJ genes, while TRA diversity relies on the m |
374) entified a number of wheat FHB resistance genes, a deeper understanding of the mecha |
375) programs corregulated with the associated genes.RESULTSOver a median (IQR) follow-up |
376) candidate subtype-specific vulnerability genes across the two tumour types confirme |
377) candidate subtype-specific vulnerability genes across three of four established sub |
378) PCs and predicts differentially expressed genes between the two lineages. |
379) in obese children, and differences in VF genes between these two cohorts. |
380) years, acquire new antibiotic resistance genes by horizontal gene transfer, and ini |
381) ng pathway, which reduces hepatic gluconeogenesis by inhibiting the gene expression |
382) expression level of IL-1β and MCP-1, two genes contributing to MMPs expression. |
383) he master regulator for the expression of genes contributing to the bacterial infect |
384) Trait Loci hotspot for a wider network of genes enriched for SCFD1 function and Amyo |
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