385) ion, and cell counting), pro-inflammatory response evaluation (ELISA for GM-CSF, IL- |
386) This consensus paper has used the human response evaluation criteria in solid tumo |
387) ive stress and pro-inflammatory cytokines response following acute myocardial infarc |
388) uable tools for studying the inflammatory response following myocardial infarction b |
389) insensitive to nonelastic response from incompressibility, but that |
390) ation strength required to evoke a muscle response from the primary motor cortex (M1 |
391) tressors that induce a systemic genotoxic response including a widely studied tumor |
392) Physiological responses, including potentiated startle, |
393) ed across these stations and expressed in response latency and magnitude were classi |
394) Previous studies have observed evoked response latency as well as gamma band sup |
395) ax immediately triggers a host senescence response mediated by cyclin-dependent kina |
396) Negative emotional response mediated the path between illness |
397) those who drop out prematurely due to non-response, or whether drop-outs may have a |
398) ven in some patients with no radiological response or early progression. |
399) sentative household survey with 8841 (60% response rate) community residents aged 16 |
400) gitudinal data from a total of 367 (81.6% response rate) middle school students in S |
401) We compared response rates, response errors, and repre |
402) to a web-based physician survey improves response rates, response quality, and repr |
403) re to lanthanoids (Ln) elicits an adverse response such as oxidative injury of lung |
404) s can be assessed on the basis of a viral response such as viral decay rate or viral |
405) For item response theory (IRT) models, which belong |
406) equate recovery of information using item response theory methods, stable test-retes |
407) The assessment of response validity is now considered an imp |
408) One way for assessing response validity is with performance vali |
409) with hypoxia network and oxidative stress response were upregulated after the lactat |
410) y) and processing style (stimulus- versus response-focused) were manipulated. |
411) ctive process, might induce an autoimmune response with potential impairment of seme |
412) imating the sensitivity of the mechanical response with the variation of basic geome |
413) elation analysis revealed two patterns of response: (1) trees on two sites in the up |
414) e studied using the auditory steady-state response (ASSR), indexing cortical activit |
415) mpled at 10 pm and the cortisol awakening response (CAR) the following morning. |
416) or cervical cancer using the early growth response (Egr-1) promoter to control sodiu |
417) y data from the participatory disease and response (PDSR) database revealed that the |
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