63) reatment has been associated with genetic factors. |
64) y the balance of pro- and anti-angiogenic factors. |
65) as influenced by social and communication factors. |
66) t model is conducted to identify the risk factors. |
67) rentiation without the addition of growth factors. |
68) used in combination with cells or growth factors. |
69) variables and the relative importance of factors. |
70) ps between genetic and environmental risk factors. |
71) esive properties of plasma rich in growth factors. |
72) s once accounting for risk and protective factors. |
73) pertension and diabetes are the main risk factors. |
74) minotransferase level, and metabolic risk factors. |
75) ) were significant abdominal obesity risk factors. |
76) come variable from a number of predictive factors. |
77) conditioned medium, without adding growth factors. |
78) socio-environmental and socio-demographic factors. |
79) ndrocyte sheets produced the most humoral factors. |
80) for association of violence with various factors. |
81) associated with genetic and environmental factors. |
82) t of multiple interacting and co-evolving factors. |
83) ombination of cells, materials and growth factors. |
84) or the evaluation of angiogenesis-related factors. |
85) geographic prevalence and associated risk factors. |
86) -rich matrix containing functional growth factors. |
87) s active but have enhanced metabolic risk factors. |
88) he relative strength of those influencing factors. |
89) nked to Obesity was used to categorize PA factors. |
90) igrant women and to identify the relevant factors. |
91) ng behaviors and school, family, and peer factors. |
92) y in China to explore its associated risk factors. |
93) fluences are significant reoffending risk factors. |
94) raction between genetic and environmental factors. |
95) mentioned in close interaction with other factors. |
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