66) Cancer cells adapt their metabolism to sup |
67) Cancer cells that are dependent on TRAF2 a |
68) were randomly implanted with MCF-7 breast cancer cells (SCT and HCT) or received an |
69) However, how cancer cells acquire the ability to evade |
70) and hormone-independent MDA-MB-231 breast cancer cells after 24 and 48 h of treatmen |
71) -neoplastic activity of emodin on bladder cancer cells and elucidates the novel mech |
72) of cultured and xenografted human breast cancer cells and prevents breast cancer de |
73) opment and the characteristic features of cancer cells are being increasingly unders |
74) ing doxorubicin (Dox) resistance in colon cancer cells are not fully understood. |
75) Altogether, we demonstrated that breast cancer cells are sensitive to the chemothe |
76) es (AuNPs) targeting CD44(+) human breast cancer cells by conventional and by propos |
77) In addition, MS analyses from pancreatic cancer cells demonstrate that lactate dehy |
78) erine 11 is essential for the survival of cancer cells harboring TRAF2 amplification |
79) Suppression of TRAF2 in cancer cells harboring TRAF2 copy number g |
80) between miRNA and Dox resistance in colon cancer cells has not been previously explo |
81) e, to investigate the behaviour of breast cancer cells in response to different envi |
82) -coated magnetic nanoparticle clusters in cancer cells in vitro and PT switching of |
83) on and invasion properties of endometrial cancer cells in vitro and their metastatic |
84) cal responses on c-MET-expressing ovarian cancer cells including increase of cell pr |
85) CDC37 overexpression in human colon cancer cells increased CDK4 protein levels |
86) In vitro cytotoxicity studies on SK-MEL-5 cancer cells indicated that 5-FU in optimi |
87) sts that the glutaminolysis in pancreatic cancer cells is different from that observ |
88) The oxidative stress in pancreatic cancer cells is lower than that in normal |
89) y, the release of oxidized DNA from dying cancer cells may give a boost to remaining |
90) of biochemical heterogeneity in mammalian cancer cells on the scale of subcellular f |
91) r in combination, had no effect on breast cancer cells sensitivity towards dox. |
92) The MS analyses from pancreatic cancer cells support a hypothesis that hyp |
93) an affect drug sensitivity of endometrial cancer cells to carboplatin. |
94) n early response of major types of breast cancer cells to Δ2-TGZ, prior to, but not |
95) Oversecretion of MUC1 provide cancer cells with invasiveness, metastasis |
96) We found that, in lung cancer cells xenograft tumors IL-7/IL-7 re |
97) en IL-7 and the lymphangiogenesis in lung cancer cells xenograft tumors, we investig |
98) specifically in sorted tumor-originating cancer cells. |
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