1) al dysfunction being the primary cause of cell death after Ag exposure under these c |
2) dynamic BH3 profiling (DBP) and apoptotic cell death assay. |
3) hways that link microtubule disruption to cell death is critical for optimal use of |
4) Tumoral programmed cell death ligand 1 (PD-L1) has been impli |
5) , where PL treatment regulated neutrophil cell death modalities including NET format |
6) , of the cytoplasmic membrane is a common cell death pathway in bacteria occurring i |
7) Because necroptosis is a cell death pathway that induces inflammati |
8) When treated with anti-programmed cell death protein 1 (PD-1) Abs, however, |
9) interferon response, including programmed cell death protein 1 (PD-L1) that is depen |
10) lls in which the expression of programmed cell death protein 1 (PD1) is downregulate |
11) the checkpoint inhibitor anti-programmed cell death protein-1 (α-PD-1). |
12) Signals specifically associated with cell death were identified by pre-treatmen |
13) plicated in excitotoxic/ischemic neuronal cell death. |
14) stent with mitochondrial toxicity causing cell death. |
15) eading to impaired cellular functions and cell death. |
16) RI-related damage that can each result in cell death. |
17) ber and a required molecule for apoptotic cell death. |
18) e, with a more severe viral infection and cell death. |
19) is required for Bak to initiate apoptotic cell death. |
20) , microvascular damage, and eventually to cell death. |
21) ion for possible contribution to neuronal cell death. |
22) in leukemic cells subsequently leading to cell death. |
23) ent androgen signaling inhibition and PCa cell death. |
24) Cell lines from this subset expressed unde |
25) Cell lines that escaped such genetic and e |
26) different RNA species from SARS-CoV-2 in cell lines and FFPE (formalin fixation and |
27) ective cytotoxic potential against cancer cell lines and good predictions for ADMET |
28) IRF8 enhancer elements that we define in cell lines and in patient samples. |
29) Rodent neuronal cell lines and primary rabbit neuronal cel |
30) d that knockdown of GRP78 in EBV-infected cell lines did not substantially affect ly |
31) ession of genes and proteins in human CRC cell lines following exposure to T3s. |
32) ies in humans, animal models, and various cell lines have revealed the role of lncRN |
33) engineered heart tissues made from these cell lines recapitulate contractile dysfun |
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