| 1) al dysfunction being the primary cause of cell death after Ag exposure under these c |
| 2) dynamic BH3 profiling (DBP) and apoptotic cell death assay. |
| 3) hways that link microtubule disruption to cell death is critical for optimal use of |
| 4) Tumoral programmed cell death ligand 1 (PD-L1) has been impli |
| 5) , where PL treatment regulated neutrophil cell death modalities including NET format |
| 6) , of the cytoplasmic membrane is a common cell death pathway in bacteria occurring i |
| 7) Because necroptosis is a cell death pathway that induces inflammati |
| 8) When treated with anti-programmed cell death protein 1 (PD-1) Abs, however, |
| 9) interferon response, including programmed cell death protein 1 (PD-L1) that is depen |
| 10) lls in which the expression of programmed cell death protein 1 (PD1) is downregulate |
| 11) the checkpoint inhibitor anti-programmed cell death protein-1 (α-PD-1). |
| 12) Signals specifically associated with cell death were identified by pre-treatmen |
| 13) plicated in excitotoxic/ischemic neuronal cell death. |
| 14) stent with mitochondrial toxicity causing cell death. |
| 15) eading to impaired cellular functions and cell death. |
| 16) RI-related damage that can each result in cell death. |
| 17) ber and a required molecule for apoptotic cell death. |
| 18) e, with a more severe viral infection and cell death. |
| 19) is required for Bak to initiate apoptotic cell death. |
| 20) , microvascular damage, and eventually to cell death. |
| 21) ion for possible contribution to neuronal cell death. |
| 22) in leukemic cells subsequently leading to cell death. |
| 23) ent androgen signaling inhibition and PCa cell death. |
| 24) Cell lines from this subset expressed unde |
| 25) Cell lines that escaped such genetic and e |
| 26) different RNA species from SARS-CoV-2 in cell lines and FFPE (formalin fixation and |
| 27) ective cytotoxic potential against cancer cell lines and good predictions for ADMET |
| 28) IRF8 enhancer elements that we define in cell lines and in patient samples. |
| 29) Rodent neuronal cell lines and primary rabbit neuronal cel |
| 30) d that knockdown of GRP78 in EBV-infected cell lines did not substantially affect ly |
| 31) ession of genes and proteins in human CRC cell lines following exposure to T3s. |
| 32) ies in humans, animal models, and various cell lines have revealed the role of lncRN |
| 33) engineered heart tissues made from these cell lines recapitulate contractile dysfun |
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