209) ypoxia-induced factor-1α and endothelial cell markers (CD31 and CD34) for the immun |
210) solated MenSCs exhibited mesenchymal stem cell markers in parallel to OCT-4 as an em |
211) ge in the absence of unique skeletal stem cell markers. |
212) e coordinated expression of downstream β-cell markers. |
213) nation, mitotic index (MI), proliferating cell nuclear antigen labeling, proliferati |
214) nation, mitotic index (MI), proliferating cell nuclear antigen labeling, proliferati |
215) to reconstruct bovine embryos by somatic cell nuclear transfer (SCNT). |
216) Interspecies somatic cell nuclear transfer (iSCNT), a powerful |
217) ation of cardiac ATDPCs caused changes in cell phenotype and genetic machinery, maki |
218) No differences in cell phenotype between the scaffold materi |
219) To sustain the β-cell-like phenotype and investigate the ef |
220) c differentiation in hAFSCs and that a β-cell-like phenotype can be augmented by cu |
221) cells recruited by O-hMSCs were the major cell populations in newly formed bone tiss |
222) Co-culture with each of the mature cell populations was shown to successfully |
223) project to various frontal areas, and all cell populations were combined in the same |
224) erentiation in co-culture with the mature cell populations. |
225) Cell-based therapies using skeletal stem c |
226) ervertebral disc to the latest molecular, cell-based therapies and tissue-engineerin |
227) The use of cell-based therapies for spinal cord injur |
228) o be an attractive and feasible source of cell-based therapies in regenerative medic |
229) Cell adhesion, morphological alterations, |
230) Cell adhesion, viability and proliferation |
231) nasal septal chondrocytes with respect to cell adhesion, migration into the matrix a |
232) Cell division kinetics revealed a higher p |
233) chemicals showed an inhibitory effect on cell division in root tips of A. |
234) mmunofluorescent double-labeling with the cell division marker Ki-67. |
235) idually or in combination on the level of cell energy (adenosine-5'-triphosphate) an |
236) ect of 2-DG and malonate by depleting the cell energy by their protective effects ag |
237) lycogen points leads to the exhaustion of cell energy that is of high demand for fis |
238) resulted in a significantly lower CD34(+) cell expansion (25-fold vs 60-, 64- and 92 |
239) igher efficiency and simpler operation of cell expansion than the PDMAAm hydrogel. |
240) Reducing cell expansion time will result in faster |
241) d HUV did not severely affect endothelial cell growth, cells seeded on decellularize |
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