243) tical downstream target of MEN1-dependent tumor suppression and is required for tumo |
244) concentration, the DA group showed a 95% tumor suppression rate without any recurre |
245) After 12 d of treatment, the tumor suppression rates of 75% and 84% wer |
246) t or senescence, to exert its function in tumor suppression. |
247) r, studies have also reported its role in tumor suppression. |
248) , which contributes to the role of p53 in tumor suppression. |
249) ession is significantly down-regulated in tumor tissue and CRC cell lines compared w |
250) on, short half-life, low concentration in tumor tissue and systemic toxicity. |
251) , 62 pairs of samples from patients whose tumor tissue showed hypermethylation of AN |
252) d and could be accumulated effectively in tumor tissue via the enhanced permeability |
253) rate was accumulated more effectively in tumor tissue. |
254) A small amount of tumor-like tissue was observed on the wall |
255) A tumor with a high level of Ki67 should be |
256) that surgical specimen displayed a solid tumor with both CLL/SLL and MCC components |
257) d strong FDG uptake (SUVmax, 34.5) of the tumor with ipsilateral hilar lymph node me |
258) Histologically, a tumor with islands of oncocytic epithelial |
259) Surgical resection of the tumor with partial resection of phrenic an |
260) ial diagnosis included primary pancreatic tumor with small cell morphology as well a |
261) Tumor angiogenesis is essential for tumor |
262) k between VEGF and MTA1 protein regulates tumor angiogenesis and metastasis. |
263) NAs (miRNAs) were important regulators of tumor angiogenesis and the entire research |
264) ecific miRNAs intersect with and modulate tumor angiogenesis is still unclear. |
265) e stochastic 3D model & simulation of tumor-induced angiogenesis. |
266) l the potential mechanism of lidamycin on tumor initiating cells and the benefit for |
267) Recently, tumor initiating cells are considered as t |
268) evaluate the effect of lidamycin on Huh7 tumor initiating cells in vitro. |
269) , we evaluated the effect of lidamycin on tumor initiating cells of hepatocellular c |
270) ts suggest that lidamycin suppressed Huh7 tumor initiating cells via GSK3β/β-caten |
271) In addition to loss of tumor suppressive function, tumor-associat |
272) that induction of apoptosis is a key p53 tumor suppressive function. |
273) owever, indicate that other undefined p53 tumor suppressive functions are operative |
274) retinoblastoma (Rb) and p53 have critical tumor suppressive functions in retinoblast |
275) enesis, and identify essential Rb and p53 tumor suppressive functions in vivo. |
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