274) transitions, how they are disrupted by T-cell interactions with tumors and the tumo |
275) tb -host cell interactions and infection outcomes i |
276) ation in velocity within the electrolytic cell is determined and validated experimen |
277) However, whether the metabolism of immune cells is dysregulated in the TME by cell-i |
278) ntigen recognition of antibodies with the cell killing properties of chemotherapy fo |
279) targets that could allow cancer-specific cell killing. |
280) eting mTOR signaling in B-ALL and other B-cell malignancies. |
281) as a common feature in a broad range of B cell malignancies. |
282) netically manipulate granule neurons in a cell-autonomous manner to study the role o |
283) nutrient partitioning was programmed in a cell-intrinsic manner through mTORC1 signa |
284) ells containing the alveolar type 2 (AT2) cell marker gene (SFTPC) and the alveolar |
285) ltiplexed RNA FISH HCR for SARS-CoV-2 and cell-type-specific marker genes. |
286) we examined blastocyst morphometry, inner cell mass and trophectoderm cell allocatio |
287) Embryo diameter, inner cell mass and trophectoderm cell number, a |
288) s irradiation had a significant impact on cell migration, improving wound healing af |
289) n to inhibit pulmonary artery endothelial cell migration, proliferation and survival |
290) ever, transcript abundances (TA) in these cell mixture samples are confounded by pro |
291) gle cell-type (B lymphocytes) directly in cell mixture samples of peripheral blood ( |
292) ted functions in interphase, specifically cell morphology and motility. |
293) of coagulation, automated hematology, and cell morphology in COVID-19, there are gap |
294) functions were generated through somatic cell nuclear transfer (SCNT). |
295) available, efficient option over somatic-cell nuclear transfer, but requires that c |
296) ism, induced dose-dependent inhibition of cell number accumulation with apoptosis as |
297) ctive aging, including reductions in stem cell number and activity, slowing meiotic |
298) r inflammatory diseases associated with T cell over-activation, exhaustion, and prem |
299) trate a sequential cellular paradigm of T cell over-activation, proliferation, exhau |
300) These enhancements were driven by greater cell permeability rather than improvements |
301) mechanoporation, which lead to increased cell permeability. |
302) going chemotherapy or haematopoietic stem cell transplant (HSCT). |
303) protocol to identify haematopoietic stem cell transplant patients at high and low r |
304) The International Society for Cell & Gene Therapy mesenchymal stroma |
305) different quantities of circulating tumor cells (CTCs) and cell-free DNA (cfDNA) int |
306) Endothelial cell (EC) sensing of wall fluid shear stre |
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