294) athway was detected by Western blot and T cell factors transcriptional activity assa |
295) transcriptional activity of β-catenin/T cell factors was decreased. |
296) erentiation medium, expression of cardiac cell-specific genes and beating cardiomyoc |
297) d EGF induced EnSCs to express urothelial cell-specific genes and proteins. |
298) Stem cell interactions through paracrine cell s |
299) y (SEM) and histology were used to detect cell-matrix interactions. |
300) falin significantly reduced serum-induced cell invasion and migration. |
301) -9, plays a key role in 5-azadC-enhanced cell invasion. |
302) was 55 days for aggressive and indolent T-cell lymphoma, 200 and 256 days for indole |
303) of 26 cases of suspect canine small-clear cell lymphoma, determined by multi-colour |
304) However, cell-seeded matrices have been recommended |
305) ograms showed that animals implanted with cell-seeded matrices maintained a wide ure |
306) Cell numbers increased to approximately 10 |
307) However, the low cell numbers available per UCB unit signif |
308) ostic disagreement, including a change in cell of origin or a change from benign to |
309) ated to different evolutive stages of the cell of origin. |
310) e using techniques such as hydrogel-based cell printing. |
311) rogels to be also used as a matrix for 3D cell printing. |
312) this study provides evidence that neural cell responses depend on the nature of the |
313) Most of the studies on cell responses towards laminin have focuse |
314) ifications in order to support successful cell seeding. |
315) ability and functionality of two advanced cell-plus-matrix seeding technologies, spr |
316) membrane and is involved in pancreatic β cell survival and function, even enhancing |
317) ion would lead to decreased neuroblastoma cell survival. |
318) nesulfonate and acridine in an eukaryotic cell system Saccharomyces cerevisiae and s |
319) We employed as model cell system the human neuroblastoma cell l |
320) ultistep development of becoming a cancer cell that allow them to survive, prolifera |
321) here is no unique master mesenchymal stem cell that is appropriate to treat all targ |
322) ards the employment of such a vehicle for cell therapy, this study examined the capa |
323) To fulfil the clinical potentials for cell-based therapy, banks of hESC lines th |
324) The responses of normal and tumor cell to oxidized DNA may differ. |
325) The specific targeting of cells to sites of tissue damage in vivo is |
326) igh-dose chemotherapy and autologous stem cell transplantation, and the subsequent i |
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