323) physiology, pathophysiology, and disease treatment as they relate to gynecologic ma |
324) mbiont-free strain obtained by antibiotic treatment as well as genome analyses revea |
325) rofile followed by 2 weeks of either Epi treatment at 1 mg/kg/day by gavage (n = |
326) veloped for evaluating only one candidate treatment at a time, and are thus not opti |
327) he Bayesian posterior probability of each treatment being optimal. |
328) HNC) are diagnosed worldwide with primary treatment being surgery and radiotherapy. |
329) The average difference in post-treatment compared to pre-treatment non-HD |
330) high Drug Burden Index (DBI) polypharmacy treatment compared to control on physical |
331) on administration, hygiene and isolation, treatment decisions, or invasive procedure |
332) To improve dengue triage and treatment decisions, the WHO recommends cl |
333) aying reward responsiveness to accelerate treatment development for neuropsychiatric |
334) ions and laboratory animals, which hamper treatment development. |
335) Moreover, statin treatment did not reduce the number of flo |
336) Importantly, statin treatment did not reverse d-flow-regulated |
337) Treatment dose (amount of treatment) has b |
338) or post-stroke aphasia and to explore how treatment dose is conceptualized, measured |
339) approach to alleviate liver fibrosis, and treatment efficacy can be monitored by obs |
340) ould very likely be necessary to maintain treatment efficacy. |
341) The use of specific clinical treatment goals (e.g., two-hour pain free |
342) iol doses ≥ 1.5 mcg/day to meet current treatment goals. |
343) ificant main effect in the high vitamin D treatment group in VDAART. |
344) Studies were included if they had a treatment group with a mean age of 65 year |
345) ties are different or the sample sizes of treatment groups are unbalanced. |
346) ion of unbalanced sample sizes in the two treatment groups or large treatment effect |
347) entlessly for the single most efficacious treatment may indirectly diminish the numb |
348) lts suggest that long-term PARP inhibitor treatment may prime both BRCA1 mutant and |
349) is that readers may focus overly on what treatment ranks best and focus insufficien |
350) or to compare the intervals or ranges of treatment ranks. |
351) Mechanistically, adropin treatment reduced BBB damage, degradation |
352) No studies showed that antifungal treatment reduced mortality. |
353) m regulator of cell fate, plasticity, and treatment resistance in NEPC that can rev |
354) etect rare subclones possibly involved in treatment resistance. |
355) tential to help in prevention, diagnosis, treatment, screening, management, and cont |
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