337) rvations and the implications for genetic risk prediction. |
338) ersely associated with a pediatric asthma risk stratification based on multiple peri |
339) nfirm causality, use of this finding as a risk stratification biomarker is promising |
340) tatus could be utilized as a predictor of risk stratification for recurrence and to |
341) tokine biomarker signatures might improve risk stratification in LTBI. |
342) Accurate detection and risk stratification of latent tuberculosis |
343) by these characteristics could facilitate risk stratification or new therapeutic tar |
344) dementia screening battery to identify at-risk individuals with DS in primary care s |
345) s of the disease, as well as detecting at-risk individuals. |
346) ' S309-CAR-NK cells for treatment in high-risk individuals as well as provide an alt |
347) media were significantly enriched in high-risk individuals. |
348) implant interface in all groups, only low-risk individuals exhibited suppression of |
349) 246 AD risk genes have not been identified as AD |
350) We identified 342 putative AD risk genes in 203 risk regions spanning 50 |
351) for developing therapeutics targeting AD risk genes or risk variants to influence A |
352) -depth functional analyses showed that AD risk genes were overrepresented in AD-rela |
353) erging from the literature, recognises at-risk populations and highlights opportunit |
354) The majority were from low-risk populations. |
355) -2019) acute/early infections in three at risk populations - MSM, high risk women (H |
356) ing in a greater number of vulnerable and risk populations of tuberculosis. |
357) hesized that a population-level polygenic risk score (PRS) can explain phenotypic va |
358) ed the predictive accuracy of a polygenic risk score (PRS) derived from a European a |
359) In the second stage, we use the baseline risk score from the first stage as a singl |
360) We find that the baseline risk score modifies the relative and absol |
361) Cardiac patients are at an increased risk to develop a severe illness if infect |
362) ressor exposures and personal factors and risk to foster methods for occupational cu |
363) by low bone mineral density and increased risk to osteoporotic fractures. |
364) included a plan to mitigate psychological risk to the researcher-suggesting a need f |
365) anders, with the aim of identifying novel risk variants associated with asthma susce |
366) Moreover, the risk variants underlying GWAS AD-associati |
367) r predicting both disease genes and their risk variants. |
368) se comparisons can identify the universal risk variants. |
369) associated with increasing numbers of at-risk alleles. |
|