386) small, reflecting a high burden of fetal growth restriction and preterm birth. |
387) sociated with high incidence of childhood growth stunting in low-income countries. |
388) rient deficiency is an important cause of growth stunting. |
389) w/v, but it did not influence follicular growth to form the pseudoantrum (60-80%; p |
390) ict and civil unrest, to rapid population growth, to combating the HIV epidemic and |
391) idence to support the importance of early growth velocities and greater degrees of w |
392) dinary least squares regression analyses, growth velocities in month 1 and individua |
393) this concentration, inhibitions on yeast growth were observed at increasing concent |
394) t of puberty, although no changes in body growth were observed in either treatment g |
395) Rapid tumor growth with caruncular and forniceal conju |
396) growth with the EC50 (concentration for 50 |
397) bventricular zone (SVZ) shows substantial growth, (2) the inner SVZ contains an incr |
398) een TAMs and the presence of infiltrative growth (P = 0.031), low tubule formation ( |
399) Cd stress decreased growth (RGR), water content (RWC), leaf os |
400) resources at a slower rate than economic growth (relative decoupling) or have even |
401) nese version of the Revised Posttraumatic Growth Inventory for Children (PTGI-C-R) a |
402) s of infants was assessed using WHO Child Growth Standards. |
403) ents: Tanner-Whitehouse, Cambridge Infant Growth Study, and the World Health Organiz |
404) associated with tumour cell invasion and growth, TGFβ1 and TGFBR1, SERPINE-1, BMP6 |
405) haracteristics of the lesion and its slow growth, a PET/CT study was developed becau |
406) For cytotoxicity testing, relative growth activity, trypan blue exclusion, (3 |
407) wever, AMPK activation can also provide a growth advantage to tumor cells by regulat |
408) nd family APGAR (adaptation, partnership, growth, affection, and resolve) scores wer |
409) etric predictor of late neointimal tissue growth after DES implantation. |
410) d proportionate increase in inhibition of growth against Escherichia coli up to 12 |
411) ators of cellular processes, such as cell growth, apoptosis, and carcinogenesis. |
412) ent a male child with a 4-year history of growth arrest, polyuria, polydipsia, recur |
413) of Glucose-Regulated Protein 78 (GRP78), growth arrest- and DNA damage-inducible ge |
414) determined the expression pattern of the growth arrest-specific transcript 5 (GAS5) |
415) There were no differences in viral growth between CB3O and CB3M. |
416) he seed germination, reduced the seedling growth by affecting a variety of biochemic |
417) molecular circuits for dynamic control of growth-essential byproduct formation pathw |
418) This process of network growth can improve access to social resour |
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