95) ysis was required to restore normal nerve function. |
96) with depressed left ventricular systolic function. |
97) hippocampal circuit plasticity to augment function. |
98) nderwent spirometric measurements of lung function. |
99) a latent variable representing cognitive function. |
100) l region that are essential for cognitive function. |
101) efore essential for neuronal survival and function. |
102) lopment and normalisation of early luteal function. |
103) were associated with poorer sensorimotor function. |
104) garding disease progression and cognitive function. |
105) exon 7, indicating impairment of protein function. |
106) sential for the assessment of the cardiac function. |
107) stem axon regeneration and improvement in function. |
108) parameters and baseline cumulative hazard function. |
109) ), and accompanied by impaired fine motor function. |
110) l function, restenosis, and mitochondrial function. |
111) pain symptomatic relief and mantenance of function. |
112) onstrated a nonsignificant improvement in function. |
113) gnificant difference in general cognitive function. |
114) f acute PTE in patients with normal renal function. |
115) y patterns and the risk of poor cognitive function. |
116) ormalising early embryogenesis and luteal function. |
117) imation of the baseline cumulative hazard function. |
118) tance of the oral cavity as a hub for its function. |
119) epresent greater biomechanical risk under function. |
120) ons may underpin deficits in sensorimotor function. |
121) understanding the mechanism of protein's function. |
122) d regulation of normal and malignant cell function. |
123) and regeneration to maintain normal brain function. |
124) all with an impairment of global systolic function. |
125) rcted myocardium and impaired ventricular function. |
126) , worry, sustained attention, and frontal function. |
127) r side effects were observed on inner ear function. |
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